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Pathophysiologic Pathways to Preterm Birth

Price:

Free

CME Credits:

1.0

CE Credits:

1.2

NOTE: Prior to submitting this exam, please print a copy for your records. Upon submission, you will be redirected to the answer key. You must answer all three (3) of the post-test questions correctly in order to pass. If you pass, you will receive your certificate within 2-4 weeks.

Step 1: Please provide your contact information
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Step 2: Complete the post-test
1. Premature activation of the hypothalamic-pituitary-adrenal (HPA) axis is primarily initiated by which risk factors?		Maternal stress Multifetal pregnancy Fetal stress Polyhydramnios both a and c
2. Prostaglandins are directly responsible for what processes leading to preterm birth?		Cervical ripening Preterm premature rupture of membranes Uterine contractions Activation of oxytocin receptors Choices a, b and c
3. Placental abruption results in the activation of:		The inflammatory pathway Thrombin production Increased corticotropin-releasing hormone (CRH) production Activation of the HPA axis None of the above
4. The following statement(s) are true:		Localized genitourinary infections can cause preterm birth Systemic infection such as periodontal disease may cause preterm birth Inflammation causes an increase in placental estrogen production Both a and b All of the above
5. The epidemiological profile of women at risk for preterm delivery overlaps that of:		Women at risk for placental abruption Women at risk for having multifetal pregnancies Women at risk for acquiring sexually transmitted infections Women at risk for polyhydramnios None of the above
6. The most common lower genital tract infection in women of reproductive age associated with preterm birth is:		T. vaginalis Candidaisis C. trachomatis Bacterial vaginosis both a and c
7. Parturition is induced by CRH through these two mechanisms:		Increase in cytokine and tumor necrosis factor a Increase tissue factor and thrombin production Increase in interleukin 1� and tumor necrosis factor a Proinflammatory maternal factors and anti-inflammatory maternal factors Increase in maternal ACTH and fetal DHEAS production
8. The condition with the highest identifiable risk for PPROM is:		Vaginal bleeding in more than one trimester Maternal cigarette smoking Maternal cocaine use Maternal hypertension with superimposed pre-eclampsia Placental pathology
Step 3: Complete feedback questions
On a scale from 1 to 5, with 5 being best, please rate how well the activity met the stated objective
Demonstrate four major pathophysiologic pathways involved in the progression to preterm delivery		5 4 3 2 1
Identify the common final pathway to preterm birth		5 4 3 2 1
Describe possible areas for research related to the pathways		5 4 3 2 1
On a scale from 1 to 5, with 5 being best, please rate the following:
Effectiveness of teaching/learning methods		5 4 3 2 1
Relevancy of this topic to clinical practice		5 4 3 2 1
Increased understanding of the topic		5 4 3 2 1
Relevancy of content to stated learning objectives		5 4 3 2 1
Please comment on the scientific rigor, fairness and balance of the content of this activity
State any changes you will make in your clinical practice as a result of this publication:
What did you like the most about this activity?
What did you like the least about this activity?
What topics would you suggest for future programs?

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